Welcome to Supportive Cancer Care Victoria (SCCV) project website developed to ensure easy access to information and resources and to foster communication and collaboration between the nine Integrated Cancer Services (ICS) in Victoria.

The SCCV project has been initiated to assist the ICS implement the Victorian Government’s supportive care policy: Providing optimal cancer care: Supportive care policy for Victoria. This policy underpins the work of the ICS to achieve high quality supportive care services for people affected by cancer and provides a framework for achieving Victoria’s Cancer Action Plan 2008-2011.

Victoria’s Cancer Action Plan 2008-2011 (VCAP) sets the direction and targets for the cancer reforms over the next three years. Action Area four of VCAP commits to strengthening supportive care services for people with cancer and their carers and establishes the following supportive care targets:

The SCCV project will assist the ICS to develop local strategies to help improve supportive care outcomes for people affected by cancer and achieve the VCAP targets.

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  • Search by: date advanced patent search home / patents / patent number 056862881 overview description abstract a novel gene, huntingtin, is described, encoding huntingtin protein, recombinant vectors and hosts capable of expressing huntingtin. Methods for the diagnosis and treatment of huntington's disease are also provided. Inventors: marcy e. Macdonald, christine m. Ambrose, mabel p. Duyao james f. Gusella original assignee: the general hospital corporation field of the invention the invention is in the field of the detection and treatment of genetic diseases. buy generic viagra online overnight Specifically, the invention is directed to the huntingtin gene (also called the it15 gene), huntingtin protein encoded by such gene, and the use of this gene and protein in assays (1) for the detection of a predisposition to develop huntington's disease, (2) for the diagnosis of huntington's disease (3) for the treatment of huntington's disease, and (4) for monitoring the course of treatment of such treatment. Background of the invention huntington's disease (hd) is a progressive neurodegenerative disorder characterized by motor disturbance, cognitive loss and psychiatric manifestations (martin and gusella, n. Engl. J. Med. Buy generic viagra 315:1267-1276 (1986). It is inherited in an autosomal dominant fashion, and affects about 1/10,000 individuals in most populations of european origin (harper, p. S. lowest price generic viagra Et al. , in huntington's disease, w. B. Saunders, philadelphia, 1991). The hallmark of hd is a distinctive choreic movement disorder that typically has a subtle, insidious onset in the fourth to fifth decade of life and gradually worsens over a course of 10 to 20 years until death. Occasionally, hd is expressed in juveniles typically manifesting with more severe symptoms including rigidity and a more rapid course. generic viagra for sale Juvenile onset of hd is associatetl with a preponderance of paternal transmission of the disease allele. The neuropathology of hd also displays a distinctive pattern, with selective loss of neurons that is most severe in the caudate and putamen regions of the brain. The biochemical basis for neuronal death in hd has not yet been explained, and there is consequently no treatment effective in delaying or preventing the onset and progression of this devastating disorder. The genetic defect causing hd was assigned to chromosome 4 in 1983 in one of the first successes of linkage analysis using polymorphic dna markers in man (gusella et al. , nature 306:234-238 (1983). Since that time, we have pursued a location cloning approach to isolating and characterizing the hd gene based on progressively refining its localization (gusella, faseb j. 3:2036-2041 (1989); guseila, adv. Hum. Genet. 20:125-151 (1991)). Among other work, this has involved the generation of new genetic markers in the region by a number of techniques (pohl et al. , nucleic acids res. 16:9185-9198 (1988); whaley et al. , somat. cheap price viagra Cell. Mol. Genet. 17:83-91 (1991); macdonald et al. , j. Clin. generic viagra buy online Inv. 84:1013-1016 (1989)), the establishment of genetic (macdonald et al. , neuron 3:183-190 (1989); allitto et al. , genomics 9:104-112 (1991)) and physical maps of the implicated regions (bucan et al. , genomics 6:1-15 (1990); bates et al. , nature genet. 1:180-187 (1992); doucette-stamm et al. , somat. order generic viagra Cell mol. Genet. 17:471-480 (1991); altherr et al. , genomics 13:1040-1046 (1992)), the cloning of the 4p telomere of an hd chromosome in a yac clone (bates et al. , am. J. Hum. Genet. generic viagra prices 46:762-775 (1990); youngman et al. , genomics 14:350-356 (1992)), the establishment of yac ›yeast artificial chromosome! (bates et al. Buy viagra online , nature genet. Cheap viagra no prescription 1:180-187 (1992)) and cosmid contigs (a series of overlapping clones which together form a whole sequence) of the candidate region, as well as the analysis and characterization of a number of candidate genes from the region (thompson et al. , genomics 11:1133-1142 (1991); taylor et al. , nature genet. 2:223-227 (1992); ambrose et al. , hum. order viagra online Mol. Genet. 1:697-703 (1992)). Analysis of recombination events in hd kindreds has identified a candidate region of 2. order generic viagra 2 mb, between d4s10 and d4s98 in 4pl6. 3, as the most likely position of the hd gene (macdonald et al. , neuron 3:183-190 (1989); bates et al. , am. order generic viagra J. Hum. Genet. 49:7-16 (1991); snell et al. , am. J. Hum. Genet. 51:357-362 (1992)). Investigations of linkage disequilibrium between hd and dna markers in 4p16. 3 (snell et al. , j. buy now online viagra Med. Genet. 26:673-675 (1989); theilman et al. , j. Med. Genet. 26:676-681 (1989)) have suggested that multiple mutations have occurred to cause the disorder (mcdonald et al. , am. J. Hum. Genet. 49:723-734 (1991)). However, haplotype analysis using multi-allele markers has indicated that at least 1/3 of hd chromosomes are ancestrally related (macdonald et al. order generic viagra , nature genet. 1:99-103 (1992)). The haplotype shared by these hd chromosomes points to a d kb segment between d4s180 and d4s182 as the most likely site of the genetic defect. Targeting this d kb region for saturation with gene transcripts, exon amplification has been used as a rapid method for obtaining candidate coding sequences (buckler et al. , proc. Natl. generic order viagra Acad. Sci. Usa 88:4005-4009 (1991)). This strategy has previously identified three genes: the a-adducin gene (adda) (taylor et al. , nature genet. discount buy viagra 2:223-227 (1992)); a putative novel transporter gene (it10c3) in the distal portion of this segment; and a novel g protein-coupled receptor kinase gene (it11) in the central portion (ambrose et al. , hum. Mol. Genet. 1:697-703 (1992)). However, no defects implicating any of these genes as the hd locu.

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Victorian State Government's Department of Health in support of Victoria's Supportive Care Policy.